Cellular mechanisms of increased skeletal muscle fatiguability in heart failure
- Prosjektnummer
- 2006172
- Ansvarlig person
- Per Kristian Lunde
- Institusjon
- Ullevål universitetssykehus HF
- Prosjektkategori
- Fritt forskningsprosjekt
- Helsekategori
- Cardiac & Cardiovaskular Systems
- Forskningsaktivitet
- Frie prosjekter - basalforskning
Increased skeletal muscle fatigability in heart failure is counteracted by an increase in calcium sensitivity of the contractile
Acta Physiologica Sinica (2008) 60.1, p191
Increased skeletal muscle fatigability in heart failure is counteracted by an increase in calcium sensitivity of the contractile
Annual meeting in Center for heart failure research, Oslo 2008
Use of single skinned muscle fibres to study the mechanism of increased skeletal muscle fatigability in heart failure.
4th Annual CHFR Symposium, Oslo, 12.oct. 2006.
Calcium sensitivity of actin-myosin force development in skeletal muscle in CHF.
5th Annual CHFR Symposium, Oslo, 11-12 oct 2007
Reduced Ca2+-activated force explains increased skeletal muscle fatigability in heart failure.
Australian Physiological Society and Australian Biophysical Society joint meeting, Newcastle, Australia 2-5 December 2007.
eRapport er utarbeidet av Sølvi Lerfald og Reidar Thorstensen, Regionalt kompetansesenter for klinisk forskning, Helse Vest RHF, og videreutvikles av de fire RHF-ene i fellesskap, med støtte fra Helse Vest IKT
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