G-protein coupled receptors in hypertension
- Prosjektnummer
- 911186
- Ansvarlig person
- Øyvind Brune Vågnes
- Institusjon
- Universitetet i Bergen
- Prosjektkategori
- Forskerutdanning - postdoc
- Helsekategori
- Urology & Nephrology
- Forskningsaktivitet
- Klinisk forskning
Short-term ANG II produces renal vasoconstriction independent of TP receptor activation and TxA2/isoprostane production.
Am J Physiol Renal Physiol 2007 Sep;293(3):F860-7. Epub 2007 jun 13
PMID: 17567934
Angiotensin II-induced calcium signaling in the afferent arteriole from rats with two-kidney, one-clip hypertension.
Am J Physiol Renal Physiol 2006 Jul;291(1):F140-7. Epub 2006 feb 7
PMID: 16467128
Prevention of hypertension and organ damage in 2-kidney, 1-clip rats by tetradecylthioacetic acid.
Hypertension 2006 Sep;48(3):460-6. Epub 2006 jul 17
PMID: 16847149
Enhanced response to AVP in the interlobular artery from the spontaneously hypertensive rat.
Am J Physiol Renal Physiol 2005 May;288(5):F1023-31. Epub 2004 des 14
PMID: 15598845
Enhanced Ca2+ response to AVP in preglomerular vessels from rats with genetic hypertension during different hydration states.
Am J Physiol Renal Physiol 2005 Jun;288(6):F1249-56. Epub 2005 jan 18
PMID: 15657301
The renal vascular response to ANG II injection is reduced in the nonclipped kidney of two-kidney, one-clip hypertension.
Am J Physiol Renal Physiol 2005 Aug;289(2):F393-400. Epub 2005 mar 22
PMID: 15784843
Inhibition of COX-2 by a bioactive fatty acid,tetradecylthioacetic acid reduces blood pressure and normalizes renal blood flow
Submitted to American Journa of Physiology, Renal Physiology
Increased expression of V1a receptors in the interlobular artery from the spontaneously hypertensive rat
Scandinavian Society of Nephrology meeting, Bergen 2005
Regulation of V1a receptors in preglomerular resistance vessels from SHR
Vintermøte Norsk hypertensjonsforening, 2005, Ringebu
eRapport er utarbeidet av Sølvi Lerfald og Reidar Thorstensen, Regionalt kompetansesenter for klinisk forskning, Helse Vest RHF, og videreutvikles av de fire RHF-ene i fellesskap, med støtte fra Helse Vest IKT
Alle henvendelser rettes til Faglig rapportering, Helse Vest