The role of Lymphotoxin beta (LTB) and LTB receptor in development of salivary gland inflammation in NOD mice; an animal model for Sjögren´s syndrome
- Prosjektnummer
- 911359
- Ansvarlig person
- Anne Isine Bolstad
- Institusjon
- Universitetet i Bergen
- Prosjektkategori
- Forskningsprosjekt
- Helsekategori
- Inflammatory and Immune System
- Forskningsaktivitet
- 6. Treatment Evaluation
Blockade of lymphotoxin-beta receptor signaling reduces aspects of Sjögren's syndrome in salivary glands of non-obese diabetic mice.
Arthritis Res Ther 2009;11(1):R24. Epub 2009 feb 18
PMID: 19222863
The minor salivary gland proteome in Sjögren's syndrome.
Oral Dis 2009 Jul;15(5):342-53. Epub 2009 apr 2
PMID: 19364392
The influence of the NOD Nss1/Idd5 loci on sialadenitis and gene expression in salivary glands of congenic mice.
Arthritis Res Ther 2007 Sep;9(5):R99. Epub 2007 sep 27
PMID: 17900348
Sjögren's syndrome--a plethora of clinical and immunological phenotypes with a complex genetic background.
Ann N Y Acad Sci 2007 Jun;1108():433-47.
PMID: 17894008
Genetic association between methyl-CpG-binding protein 2 (MECP2) and primary Sjögren’s syndrome.
10th International Symposium on Sjögren´s syndrome, Oct. 2009, Brest, France.
Fcg receptor IIA, IIIA and IIIB single nucleotide polymorphisms and Fcg receptor IIIB copy number variation: No association wit
10th International Symposium on Sjögren´s syndrome, Oct. 2009, Brest, France.
Features of lymphoid neogenesis in non-obese diabetic mouse thymus.
10th International Symposium on Sjögren´s syndrome, Oct. 2009, Brest, France.
Treating Established Experimental Sjögren’s Syndrome by Blocking the Lymphotoxin Beta Receptor Pathway Improves Salivary Gland F
10th International Symposium on Sjögren´s syndrome, Oct. 2009, Brest, France.
Genes associated with Systemic Lupus Erythematosus (SLE) that are shared with Sjögren’s Syndrome (SS).
10th International Symposium on Sjögren´s syndrome, Oct. 2009, Brest, France.
Candidate genes for Sjögren’s syndrome (SS) susceptibility can be identified by genome wide scans (GWAS) with pooled DNA.
10th International Symposium on Sjögren´s syndrome, Oct. 2009, Brest, France.
Possible association of Sjögren’s Syndrome (SS) with THRB, the thyroid hormone receptor beta.
10th International Symposium on Sjögren´s syndrome, Oct. 2009, Brest, France.
Blockade of lymphotoxin-beta receptor signaling reduces aspects of Sjögren`s syndrome in salivary glands of NOD mice
Submitted - revision requested
The minor salivary gland proteome in Sjögren´s syndrome
Submitted - pending
Association of Sjögren´s syndrome (SS) and Systemic lupus erythematosus (SLE) with BLK. FOCIS 2008.
Clin Immunol. 2008;127 Suppl:S41.
Blockade of Lymphotoxin-Receptor Axis Reduces Aspects of Sjögren's Syndrome in Salivary Glands of NOD Mice.
Arthritis Rheum 2008; 57;9, ( 711), S430.
Genetic aspects of Sjögren´s syndrome
Oral Diseases. 14; Suppl I (1), 2008.
Association of BLK with Sjögren's Syndrome (SS) and Systemic Lupus Erythematosus (SLE) initially identified by whole genome DNA
Arthritis Rheum 2008; 57; 9, (1688), S791.
Blockade of lymphotoxin-beta receptor axis reduced HEVs and Lymphocytic infiltrates in the NOD model of Sjögren´s syndrome.
11th International TNF Conference, Asilomar, CA, USA, May 13-16, 2007.
Lymphotoxin-beta receptor axis modulates HEV status and gene expression patterns in exocrine glands in NOD mouse model of Sjögre
Arthritis Rheum 56 (9)Suppl.: S441, 1049.
Identification of differentially expressed proteins in minor salivary glands from patients with Sjögren`s syndrome and healthy c
13th International Congress of Immunology. Rio de Janeiro, Brazil, 21-25th Aug 2007. Control No. 2007-A-3494-ICI.
Signaling to nuclear factor kappa B in the lymphoid microenvironment in Sjögren´s syndrome and NOD mice
- Disputert:
- juni 2009
- Hovedveileder:
- Kathrine Skarstein/Anne Isine Bolstad/Malin Jonsson
Gene expression and protein profiling in human and experimental Sjögren´s syndrome
- Disputert:
- april 2008
- Hovedveileder:
- Anne Isine Bolstad
eRapport er utarbeidet av Sølvi Lerfald og Reidar Thorstensen, Regionalt kompetansesenter for klinisk forskning, Helse Vest RHF, og videreutvikles av de fire RHF-ene i fellesskap, med støtte fra Helse Vest IKT
Alle henvendelser rettes til Faglig rapportering, Helse Vest